GAB1 Primary Antibody

Item Information
Catalog #Size/ConcentrationPrice
Specification
AliasesN
ProductOrderG
Clone#5F11C8
Entrez GenelD2549
FormulationPurified antibody in PBS with 0.05% sodium azide
HostMouse
IsotypeIgG1
ImmunogenPurified recombinant fragment of human GAB1 (AA: 661-724) expressed in E. Coli.
MW32.4kDa
Shipping InformationThis product will ship in a box containing blue ice at a temperature of 4°C. Learn More
Species ReactivityHuman
Application
ELISA1/10000
FCM (Flow Cytometry)1/200 - 1/400
WB (Western Blot)1/500 - 1/2000
Sequence
661-724
Catalog#: 30748
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Images
Western Blot
Figure 1: Western blot analysis using GAB1 mAb against human GAB1(AA:661-724) recombinant protein. (Expected MW is 32.4 kDa)
Figure 1: Western blot analysis using GAB1 mAb against human GAB1(AA:661-724) recombinant protein. (Expected MW is 32.4 kDa)
Western Blot
Figure 2: Western blot analysis using GAB1 mAb against HEK293 (1) and GAB1 (AA:661-724)-hIgGFc transfected HEK293 (2) cell lysate.
Figure 2: Western blot analysis using GAB1 mAb against HEK293 (1) and GAB1 (AA:661-724)-hIgGFc transfected HEK293 (2) cell lysate.
Flow cytometric
Figure 4: Flow cytometric analysis of Hela cells using GAB1 mouse mAb (green) and negative control (red).
Figure 4: Flow cytometric analysis of Hela cells using GAB1 mouse mAb (green) and negative control (red).
Elisa
Black line: Control Antigen (100 ng); Purple line: Antigen(10ng); Blue line: Antigen (50 ng); Red line: Antigen (100 ng);
Black line: Control Antigen (100 ng); Purple line: Antigen(10ng); Blue line: Antigen (50 ng); Red line: Antigen (100 ng);
Product Overview
Description

The protein encoded by this gene is a member of the IRS1-like multisubstrate docking protein family. It is an important mediator of branching tubulogenesis and plays a central role in cellular growth response, transformation and apoptosis. Two transcript variants encoding different isoforms have been found for this gene.

References (references)
References (references)Cell Signal. 2009 Dec;21(12):1775-83.
Cell. 2009 Jan 23;136(2):352-63.